Mitochondria localized BCL-XL acts as an apoptosis inhibitor

Öffentlichkeit Deposited

Abstract: "HeLa cells over-expressing the BCL-XL protein were treated with two pro-apoptotic stimuli; DNA damage (UV treatment) and activation of tumor necrosis factor (TRAIL treatment)."-p.1.

19 unnumbered pages : PDF file ; cm.

Titel
  • Mitochondria localized BCL-XL acts as an apoptosis inhibitor
Zuletzt geändert
  • 06/06/2024
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  • Missouri Southern State University Archive
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  • All documents property of Missouri Southern State University. Contact [email protected] for further information.
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  • Adobe Acrobat PDF